Re: A fully developed creature can evolve?


"Perplexed in Peoria" <jimmenegay@xxxxxxxxxxxxx> wrote in message
news:dfv7s3$2g9d$1@xxxxxxxxxxxxxxxxxxxxxx
>
> "Artificer" <eliezerfigueroa@xxxxxxxxx> wrote in message
> news:dftqi4$20bh$1@xxxxxxxxxxxxxxxxxxxxxx
>> I have a curiosity!. A fully developed creature like an adult human
>> being is likely to suffer minor genetics (evolutionary) changes during
>> its live or the genetic deviation from the parents always occur during
>> de conception and formation of the creature? This question is about
>> complex creatures not for bacterial organisms!
>
> I'm not sure what you mean by "minor genetics (evolutionary) changes".
> I'm going to assume that you mean mutations.
>
> Well, the usual assumption is that a mutation happens in a single cell.
> If a mutation happens in a single cell of an adult, it will probably
> not do much for/to him. Well, if it happens in a type of cell that
> divides frequently it may have some effect - it it happens in a white
> blood cell it could conceivably confer immunity to AIDS, but more likely
> will cause leukemia.
>
> But whatever it does to the adult, it won't have an important evolutionary
> impact unless it can be passed on to the children. And it can't. Unless,
> that is, the mutation happens in a 'germ line' cell destined to become
> a sperm or egg. That can be passed on. In fact, most mutations happen in
> the parent before sex, rather than in the fertilized egg after sex. But
> such mutations in the parent probably won't have much effect on the
> parent.

Art,

There was a man in Finland, a few generations ago, who -- it has been
verified --
had a mutation in a 'germ line' cell 'destined to become a sperm, which
resulted in
an equivalent of a 'typo error' in the copying of his DNA. One letter got
substituted for another letter.

The resulting DNA change went to all his offspring and has been passed from
them to all their offspring,
etc., every generation since. The altered allele (an allele is a code
sequence in the DNA that influences certain protein foldings in such a way
as to result in observable traits) was in a zone which impacts
pulmonary auto-immune function. It was recessive (meaning that unless a
descendant got it from both parents, it did not get expressed).

Despite the fact there was not the sophistication we have today about DNA,
researchers were able to trace back to find a common relative, and to work
out mathematically where the gene came from. (Just hitting the high points
here.)

It is important to understand that, in a population there can be thousands
of people who share a common relative. This is not speculative. It is
established fact. Lots and lots of Finns are third, fourth, fifth, sixth...
cousins, due to the fact the country was relatively stable for many, many
generations.

Because of this, genetic counseling is given to most young couples before
they marry. The likelihood is very much higher there, that one or more
genetic abnormalities will occur there, because the statistical likelihood
of a couple's BOTH having the recessive gene for the particular trait traced
back to the individual mentioned above, passed on by the one individual
mentioned above, or both having some OTHER pairing of a set of some of the
more than fifty known "bad" recessive genes common in the population of
Finland, is extraordinarily high there.

More attention is given by medical and anthropological genetic researchers
to "bad" genes than to "advantageous" genes.

Hopefully that will not always be true, but so far, where there is money for
devoting to research, it has seemed more important to find out why people
with certain alleles tend to die young of cardio-vascular problems, or have
type one, or type two, diabetes, or be born with kidneys that will fail
early in life, or are mentally retarded... than to seek to identify alleles
that result in, say, genius-level IQ, or who are superstar athletes, or who
go through adolescence with no zits.

I don't know if this answers your question. It is aimed only at giving you
a glimpse of what goes on with
mutations.

In the case of three hundred years or so in Finland, the "beneficial"
mutations may not seem to explain much about how populations can accumulate
changes -- good, bad or indifferent. Multiply that by a million years, and
you begin to get a feel for how changes can add to changes which add to
changes... giving advantages to some and disadvantages to others and how...
over millions of years... these take on increasing significance.

If I have said anything here that is not exactly correct, I hope Jim will
correct it.

My only purpose is just to give you just the most cursory bit of a feel for
what things you would want to delve into, if you would like to seek a
sophisticated understanding of how the gene/mutation/advantage vs.
disadvantage 'screening' process works, over the long, long haul.

You might think of it as the kindergarten version. Start digging in and
learning, and you may find yourself spending a lot of time getting to know
the nitty of it.

Also, there is new research data pouring in from all over the civilized
world on this, and if you reach a high level of knowledge on it, and want to
stay on top of it, be prepared to work hard just reading all the voluminous
amount of new info coming out of the end of the pipeline DAILY nowadays.

g








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