Re: Question about sperm and egg?

[This is a corrected resend where the word "allometric" replaces the
incorrect word "allopatric"]

"Perplexed in Peoria" jimmenegay@xxxxxxxxxxxxx wrote:-

JE:-
After fertilization one enormously large fertilized egg cell unequally
subdivides itself into a large number of tiny cells of about the same
mass
_unequally_ dividing up the fertilized egg cytoplasm which contains
strategically placed concentrations of many different organizing
substances.
Some of these are RNA's contributed by genes from the mother's genome
and
NOT the genome of the embryo. Cytoplasmic RNA's can keep an enucleated
egg
subdividing to an early stage of development all by themselves. Many
different and varied cytoplasmic based organizing substances are
deliberately allocated in a NON equal pattern within each egg. Thus the
cytoplasmic concentrations within each tiny cell become different
whereas
the embryo DNA in most cases remains equally replicated. This provides a
"prima face" case that non nuclear and other substances control the
basic
embryo body plan allowing the possibility of critical controlling
substances
passing from mother to offspring as envisaged by Darwin's "pangene"
epigenetic hypothesis. At the very least, genes that are NOT inherited
from
the mother can still play a critical developmental role for that
mother's
embryo.

Yes indeed. It is called "maternal effect". And in man, the mother's
genes continue to have an effect on the fetus in utero. And both parents
exert a cultural effect at least until the child reaches maturity.
But the portion of the maternal effect caused by genes in the mother that
are not passed to the child will have a negligible effect on the
grandchildren.

JE:_
Yes, but each viable egg is a _complete_ egg. Every viable egg DOES contain
a heritable pattern Re: the concentration of many and varied inducing
substances which DOES set the foundation for the critical, early, more
creative morphogenic stage. Each large egg DOES subdivide itself using an
UNEQUAL form of mitosis into many tiny cells of about the same mass
transforming the distribution pattern of morphogenic induction substances
within the egg into a _cell pattern_ BEFORE the allometric stage vastly
increases embryo mass using just an EQUAL form of mitosis. No gene has been
identified which can code for a particular body plan or even just one
element within a plan. It appears that the genes within genome of the embryo
are mostly concerned with the allometric growth stage which of course
remains dependent on the earlier morphogenic stage. How/where is a heritable
morphogenic pattern stored and how does it becomes retrieved over countless
egg generations? How does one egg morphogenic body plan mutate and change
into another?

Selection can and does act on ANY form of heritable information. This
includes heritable epistatic and epigenetic information. Both actually
EXIST. Waddington's ignored-for-over-50-years correction of Haldane's gene
centric population genetics model (which still forms the basis of today's
population genetics) remains the only Neo Darwinistic MINIMALLY _organism_
centric model which can provide a selective coefficient for all the ignored
hidden epistatic genes that he EMPIRICALLY observed are required to code for
a heritable phenotype said to be coded for by just major genes. Yet, single
major genes as just single locus models remain the sole concern of Haldane's
population genetics.

The genes used within today's gene centric Haldane derived population
genetics are only CORRELATED to a particular phenotype. Such correlated
models delete the majority/all heritable epistatic information and all
heritable epigenetic information within convenient simplified/oversimplified
models without correcting for either deletion within a proposed
_generalization_ (a synthesis such as evolutionary theory) e.g. Hamilton's
Rule. Failing to correct for such drastic deletions within a proposed theory
induction constitutes a serious error. For this reason gene centric Neo
Darwinistic generalizations can only constitute a misuse of
simplified/oversimplified models of Darwinism within which the majority of
heritable information, including all empirical epistatic gene fitness,
remain deleted. The unexpected tiny size of both the human and the chimp
genomes proved that Haldane's dilemma was just an artifact caused by the
deletion of huge quantities of heritable epistatic and epigenetic
information.

Regards,

John Edser
Independent Researcher

edser@xxxxxxxxxx








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