Re: Diabetes and Potassium?



On Jan 31, 6:14 am, "Andrew B. Chung, MD/PhD"
<and...@xxxxxxxxxxxxxxxxxxx> wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor "kumar" wrote:

Hello,

In diabetes, potassium may fall after you take insulin, particularly if
your diabetes had been out of control for a while. Low potassium is
commonly due to "water pills" (diuretics).

"INSULIN Actions on cellular and metabolic level
Increased potassium uptake - forces cells to absorb serum potassium;
lack of insulin inhibits absorption.
Arterial muscle tone - forces arterial wall muscle to relax,
increasing blood flow, especially in micro arteries; lack of insulin
reduces flow by allowing these muscles to contract.http://en.wikipedia.org/wiki/Insulin";

"Bulimia
chem-20 may show an electrolyte imbalance (such as hypokalemia) or
dehydration.http://www.nlm.nih.gov/medlineplus/ency/article/000341.htm";

Potassium instabilties/imbalances looks to be related to diabetes.
Increased efflux of Potassium in extracellular compartment due to
hyperglycemia and its excretion in urine, looks to be possible. Losing
more Potassium due to polyuria can also be thought. It also looks
defficieny of potassium may stimulate binge eationg..common in
diabetics.

As such, can there be some defficiency of potassium in diabetics2 esp.
in uncontrolled people pre-renal damages) and can it stimulate
excessive intake of food or binge eating resulting into overeatings?

Hypokalemia typically results in weakness, malaise, and anorexia.

Without hunger (anorexia), overeating does not happen.

Severe hypokalemia can lead to cardiac dysrhythmias that can result in
death.

While you are hungry, you know you are not dying.

Efflux of K can be there due to hyperglycemia, which may cause an
impression of hyperkalemia in short run still intracellular levels be
decreased. This efflux of K can cause excretion of more K in urine
continusly as homeostatis try to maintain normal level of K in blood
and may also stimulate more hunger for more K intake. This condition
may be called as defficience K in cells but more in ECF, in blood and
in urine supported by more K intake in food. However it may be
difficult to maintain chronically or on decreased food intake it may
cause hyperkalemia and express symptoms of hypokalemia. So both
hyperkalemia and hypokalemia can be possible in case of persisting
hyperglycema. It is important to note, many people, still with
hyperglycemia may feel symptoms of hypoglycema type, which actually be
of hypokalemia. As such, a diabetic2 with persisting hyperglycemia may
be of any type in K levels...hyperkalemic, hypokalemic, defficient in
ICF. Added insulin somewhat tries to reverse these conditions by
mediating influx of K into the cells as per above mentioned action of
insulin.

Now, if body level or cellular level of K is defficient due to
increased efflux and losing it in urine, I think it can stimulate more
hunger for K in food to compensate the loss of more K lost in urine?

Again, deficiencies of vital nutritients and minerals cause a loss of
appetite.

Is it necessary that Mg & K defficient in ICF will also show lower
than normal levels in blood?

Yes.
How?
As such, it can be thought that defficiency of any nutrient or on
excessive losing of any nutrient, excessive hunger can be there to
compensate defficiency of that nutrient?

Excessive losses of vital nutrients causes a loss of appetite as the
person is dying.

On glycosuria, Glucose is lost in diabetics with hyperglycemia, still
that person don't lose appetite?

The appetite would not be lost until there is hypoglycemia.
How body's system promote defficiency or possible defficiency of any
nutrient?

How we get hunger?
Any vital nutrient, Even lost in excess, but a person compensate by
its excessive intake, how he can be considered as dying?

When there is loss of appetite, intake stops.

It is another isuue that a person defficient in any nutrient take whole
food which may cause excess intake of other nutrients along with that
more food and cause other problems. Probably, a diabetic may be
losing/excreting or taking more of other nutrients alike K---need to be
understood. Probably such variations in K levels may also effect pH,
Na, Ca, Mg, water levels etc. indicative in short or long run. Ok?

With such loss of homeostasis, a person will be dying.

Dying people lose their appetite.

If a person can compensate by food intake, how can he be considered
dying?

With loss of appetite, a person will not be able to compensate with
food intake.

Whether urine potassium is regularily checked?

No. Not necessary.

Whether a persitant hyperglycemic diabetic lose more of K and Mg in
urine?

Not clinically seen.

"Hunger is a blessing."

Andrew <><
--
Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text -

- Show quoted text -


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