Re: Diabetes and Potassium?

On Feb 3, 9:50 am, "Kumar" <lordshiva5...@xxxxxxxxxxxxxx> wrote:
On Feb 2, 7:55 pm, "Andrew B. Chung, MD/PhD"





<and...@xxxxxxxxxxxxxxxxxxx> wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor "kumar" wrote:

Hello,

Indiabetes, potassium may fall after you take insulin, particularly if
yourdiabeteshad been out of control for a while. Low potassium is
commonly due to "water pills" (diuretics).

"INSULIN Actions on cellular and metabolic level
Increased potassium uptake - forces cells to absorb serum potassium;
lack of insulin inhibits absorption.
Arterial muscle tone - forces arterial wall muscle to relax,
increasing blood flow, especially in micro arteries; lack of insulin
reduces flow by allowing these muscles to contract.http://en.wikipedia.org/wiki/Insulin";

"Bulimia
chem-20 may show an electrolyte imbalance (such as hypokalemia) or
dehydration.http://www.nlm.nih.gov/medlineplus/ency/article/000341.htm";

Potassium instabilties/imbalances looks to be related todiabetes.
Increased efflux of Potassium in extracellular compartment due to
hyperglycemia and its excretion in urine, looks to be possible. Losing
more Potassium due to polyuria can also be thought. It also looks
defficieny of potassium may stimulate binge eationg..common in
diabetics.

As such, can there be some defficiency of potassium in diabetics2 esp.
in uncontrolled people pre-renal damages) and can it stimulate
excessive intake of food or binge eating resulting into overeatings?

Hypokalemia typically results in weakness, malaise, and anorexia.

Without hunger (anorexia), overeating does not happen.

Severe hypokalemia can lead to cardiac dysrhythmias that can result in
death.

While you are hungry, you know you are not dying.

Efflux of K can be there due to hyperglycemia, which may cause an
impression of hyperkalemia in short run still intracellular levels be
decreased. This efflux of K can cause excretion of more K in urine
continusly as homeostatis try to maintain normal level of K in blood
and may also stimulate more hunger for more K intake. This condition
may be called as defficience K in cells but more in ECF, in blood and
in urine supported by more K intake in food. However it may be
difficult to maintain chronically or on decreased food intake it may
cause hyperkalemia and express symptoms of hypokalemia. So both
hyperkalemia and hypokalemia can be possible in case of persisting
hyperglycema. It is important to note, many people, still with
hyperglycemia may feel symptoms of hypoglycema type, which actually be
of hypokalemia. As such, a diabetic2 with persisting hyperglycemia may
be of any type in K levels...hyperkalemic, hypokalemic, defficient in
ICF. Added insulin somewhat tries to reverse these conditions by
mediating influx of K into the cells as per above mentioned action of
insulin.

Now, if body level or cellular level of K is defficient due to
increased efflux and losing it in urine, I think it can stimulate more
hunger for K in food to compensate the loss of more K lost in urine?

Again, deficiencies of vital nutritients and minerals cause a loss of
appetite.

Is it necessary that Mg & K defficient in ICF will also show lower
than normal levels in blood?

Yes.

How?

The tendency for cations to diffuse from an area of higher
concentration to an area of lower concentration.

Can such diffusion of K just be effected by hypertonicity of ECF,
supoose if glucose is higher in ECF?

No.

As such, it can be thought that defficiency of any nutrient or on
excessive losing of any nutrient, excessive hunger can be there to
compensate defficiency of that nutrient?

Excessive losses of vital nutrients causes a loss of appetite as the
person is dying.

On glycosuria, Glucose is lost in diabetics with hyperglycemia, still
that person don't lose appetite?

The appetite would not be lost until there is hypoglycemia.

How body's system promote defficiency or possible defficiency of any
nutrient?

Deficiency is not promoted.

Sorry, how body system compensate used substances or possible
defficiencies?

It does not.

For this reason, it is wise to eat a varied diet in order to avoid
deficiencies while eating the optimal amount. Your hunger will inform
you that you have avoided deficiencies.

"Hunger is good." -- Holy Spirit

Amen.

Andrew <><
--
Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com-Hide quoted text -

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""What are the symptoms of hyperkalemia?

Hyperkalemia can be asymptomatic, meaning that it causes no symptoms.
Sometimes, patients with hyperkalemia report vague symptoms including
nausea, fatigue, muscle weakness, or tingling sensations. More serious
symptoms of hyperkalemia include slow heartbeat and weak pulse. Severe
hyperkalemia can result in fatal cardiac standstill (heart stoppage).
Generally, a slowly rising potassium level (such as with chronic
kidney failure) is better tolerated than an abrupt rise in potassium
levels.

What causes hyperkalemia?

The major causes of hyperkalemia are kidney dysfunction, diseases of
the adrenal gland, potassium sifting out of cells into the blood
circulation, and medications.

Kidney dysfunction

Potassium is normally excreted by the kidneys, so disorders that
decrease the function of the kidneys can result in hyperkalemia. These
include acute and chronic renal failure, glomerulonephritis, lupus
nephritis, transplant rejection, and obstructive diseases of the
urinary tract, such as urolithiasis (stones in the urinary tract).

Furthermore, patients with kidney dysfunctions are especially
sensitive to medications that can increase blood potassium levels. For
example, patients with kidney dysfunctions can develop worsening
hyperkalemia when given salt substitutes that contain potassium, when
given potassium supplements (either orally or intravenously), or
medications that can increase blood potassium levels. Examples of
medications that can increase blood potassium levels include ACE
inhibitors, nonsteroidal anti-inflammatory drugs (NSAIDs), Angiotensin
II Receptor Blockers (ARBs), and potassium sparing diuretics (see
below).

Diseases of the adrenal gland

Adrenal glands are small glands located adjacent to the kidneys, and
are important in secreting hormones such as cortisol and aldosterone.
Aldosterone causes the kidneys to retain sodium and fluid while
excreting potassium in the urine. Therefore diseases of the adrenal
gland that lead to decreased aldosterone secretion can decrease kidney
excretion of potassium, resulting in body retention of potassium, and
hence hyperkalemia.

Potassium shifts

Potassium can move out of and into cells. Our total body potassium
stores are approximately 50 mEq/kg of body weight. At any given time,
about 98% of the total potassium in the body is located inside of
cells (intracellular), with only 2% located outside of cells (in the
blood circulation and in the "extracellular" tissue). The blood tests
for measurement of potassium levels measure only the potassium that is
outside of the cells. Therefore, conditions that can cause potassium
to move out of the cells into the blood circulation can increase the
blood potassium levels even though the total amount of potassium in
the body has not changed.

One example of potassium shift causing hyperkalemia is diabetic
ketoacidosis. Insulin is vital to patients with type 1diabetes.
Without insulin, patients with type 1diabetescan develop severely
elevated blood glucose levels. Lack of insulin also causes the
breakdown of fat cells, with the release of ketones into the blood,
turning the blood acidic (hence the term ketoacidosis). The acidosis
and high glucose levels in the blood work together to cause fluid and
potassium to move out of the cells into the blood circulation.
Patients withdiabetesoften also have diminished kidney capacity to
excrete potassium into urine. The combination of potassium shift out
of cells and diminished urine potassium excretion causes
hyperkalemia.

Another cause of hyperkalemia is tissue destruction, dying cells
release potassium into the blood circulation. Examples of tissue
destruction causing hyperkalemia include trauma, burns, surgery,
hemolysis (disintegration of red blood cells), massive lysis of tumor
cells, and rhabdomyolysis (a condition involving destruction of muscle
cells).

Medications

Potassium supplements, salt substitutes that contain potassium and
other medications can cause hyperkalemia.

In normal individuals, healthy kidneys can adapt to excessive oral
intake of potassium by increasing urine ...

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Dear Kumar
I ran into your message quite accidentally while researching about
some details on ''Diabetes' 'and thought of sharing some of my
findings.
I've read at 'http://www.medical-health-care-information.com/Health-
living/Diabetes/index.asp'
that Diabetes is a set of related diseases in which the body cannot
regulate the amount of sugar (glucose) in the blood. In diabetes,
glucose in the blood cannot move into cells, and it stays in the
blood. This not only harms the cells that need the glucose for fuel,
but also harms certain organs and tissues exposed to the high glucose
levels.
I hope the above is of some help to you as well. Regards, Sherrybove

.

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