Yale, Ax- Boy and Bicycle- Boy...Re: has anyone ever done this study?

From: kathleen (kathleen.dickson_at_snet.net)
Date: 03/22/05 

Date: 22 Mar 2005 04:55:15 -0800

I am almost certain the expert in the Lyme Rage
Ax trial was Bransfield, since I was there and
attended. But not every day.

http://www.yaledailynews.com/article.asp?AID=406

I talked to the Mom. She thought it was
Suicide by Cop. A Yale-trained Forensic
Psychiatrist of course, determined the kid was sane.

Lyme is a permanent brain and nervous system
disease. He should have another trial, since
the testimony of the Yale trained Forensic
Psychiatrist was obviously biased.

Bransfield's testimony was very, very convincing.

But the judge had predecided the outcome, in
my opinion.

Everyone in the whole world thought just
jumping in your car, and randomly picking a
house to attack the resident of, was psychotic.

Resolving frustration aspect of not validating the
seriousness of this illness is central to treatment,
recovery, and rehabilitation. Validate the illness.

Chronic Fatigue, ME, and Fibromyalgia patients go
through the same thing we do.

One of the Lyme criminals calls these "catastrophizing"
and infers, that even FM and CFIDS are not real.

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=11083273

Where is the outrage? (So to speak.)

FEATURE ARTICLE from The Washingtonian, 1-91, By Neil Raven

``Bicycle Boy -- His Behavior Was Compulsive, It's Origins Unknown;
Then a Good Doctor Seemed to Make a Miracle Happen

He was 12 years old, and every day he pedaled furiously on his
stationary bicycle for as many hours as they would allow him. He was so
absorbed in his effort that it was all they could do to get him to stop
for meals.

In fact, before he was hospitalized at a psychiatric institution he had
been unwilling to stop for meals, for school work, for the simple
exchanges of ordinary life. At age 12, he had lost almost 30 pounds. He
looked, in the language of the ward, cachetic, or in the language of
his friends, as if he had been an inmate in a concentration camp.

His parents, after all the agonizing, had coaxed him into a car and
driven him out to the facility , where they had carried his suitcase as
they walked him to the ward. And they had handed their son over to the
care of others, out of desperation, convinced that he was now beyond
their help - their son who wanted only to pedal, to exert himself and
withdraw from the world he had once embraced with such sunny
exuberance.

The psychiatrists questioned the parents and the boy - the skeletal,
restless boy, who not so long before had been a good student, a
healthy, happy son. He had been a wonderful athlete, an exciting soccer
player, but he had had some knee problems. Over two years he had had
four episodes in which his right knee swelled enough to require
treatment.

It was after the last episode that he had withdrawn. He spent most of
his time alone in his room, fiddling with a ham radio, not talking to
his friends or his parents. He stopped doing his homework. And then
came the exercising, the disinterest in food, the weight loss.

At a glance, the boy reminded the psychiatrists of the young women who
suffered from that dreaded and potentially lethal psychiatric
condition, anorexia nervosa. He had that bony look, that restless
hyperactivity.

But he was male, which is unusual for anorexia nervosa patients. And he
was only 12 -- most patients with anorexia nervosa are older. It could
be a working diagnosis. But when things don't quite fit the pattern,
you ask questions. You call in more opinions. They called in Andrew
Pachner.

Andrew Pachner looks over to the framed photograph on the wall of his
office at the Georgetown university Hospital's neurology department.
The photo is a blowup of a single Borrelia burgdorferi spirochete - a
microorganism that bears a striking resemblance to the organism that
causes syphilis ...

[Dr. Pachner] recalls the day he first laid eyes on the 12-year-old
bicycle boy. Pachner was then a junior faculty member in the Department
of Neurology at the Yale School of Medicine, living on a salary that
didn't even approach subsistence level. While the university looked the
other way, all the junior faculty members moonlighted to pay the rent.
Among Pachner's stints was a job evaluating patients at the psychiatric
institute.

Not all patients were selected by the psychiatrists for Pachner's
review. But the bicycle boy was. For one thing, there were those
swelling episodes and the probable history of arthritis.

While he was still in training, Pachner had drifted down to the Yale
arthritis clinic. Diseases of the joints might seem an unlikely source
of fascination for a doctor specializing in diseases of the nervous
system, but there was a vital connection. Diseases of both are often
[sometimes?] caused by mistakes that cause the immune system to turn
against itself - autoimmune diseases.

The doctors studying arthritis were happy to have Pachner around. Many
of their arthritis patients were suffering from autoimmune diseases,
such as systemic lupus erythematosus, which have neurologic
complications. Pachner's neurology expertise was welcomed.

While Pachner was examining patients in the arthritis clinic, he became
an interested bystander to one of the most celebrated moments in
medicine - the identification of a new disease.

... a group of children in Old Lyme, Connecticut, not far from Yale ...
had a curious form of arthritis that followed the appearance of a
peculiar and characteristic skin rash called erythema chronicum
migrans, or ECM. [first described in this country by Dr. Scrimenti in
Wisconsin, in 1970.]

... In 1982, Drs. Willy Burgdorfer and Alan Barbour, working at the
Rocky Mountain Laboratory in Montana, pinpointed the cause of the
disease.

... [The] young patients had an arthritis caused by a spirochete.
Unlike bacteria, spirochetes are not easily grown in the laboratory.
The standard way to study a microorganism is to grow it on a special
broth, a culture plate.

But spirochetes, like exotic zoo animals, do not live long outside
their native habitats. Once outside the body, they die. The human body
makes antibodies to the organism , which makes diagnosis possible, but
the antibody tests can be tricky, and occasionally misleading.

The world's best-known spirochete is Treponema pallidum, which causes
syphilis. The one that causes Lyme disease would prove to be an even
bigger problem than syphilis in some ways, because people could not
avoid it by abstemious behavior.

It was a spirochete that awaited children as they ran through the
Connecticut woods, doing what their parents thought was healthy and
good. The spirochete was carried by forest animals, and it waited for
the unsuspecting, anyone who cared to enjoy the great outdoors: hikers,
pregnant women toting little kids, fishermen, gardeners, and farm
workers. It was the tick-borne spirochete that causes Lyme disease.

The bicycle boy had had his first attack of Lyme arthritis in 1982, two
years before Pachner discovered him pedaling away on the psychiatric
ward.

Pachner was aware that syndromes similar to Lyme arthritis, syndromes
suspected to be caused by an infectious agent, had been described in
Europe, and he knew these syndromes often included some neurological
features, usually a form of radicular pain , which radiates down an arm
or a leg. Radiculitis meant the trouble was in peripheral nerves, which
flow to and from the spinal cord out to the extremities.

But none of the these arthritis-related European syndromes involved the
central nervous system. None of these European syndromes caused complex
behavioral changes, and no connection had ever been drawn between an
infectious arthritis and any sort of neurological disease that might
affect a person's behavior.

In order to cause a behavioral change, a disease has to affect the
brain directly and in a widespread fashion. Various forms of vasculitis
- inflammation of the small blood vessels - can do this. Autoimmune
diseases can do this.

But none of the infectious - arthritis group of diseases were known to
be capable of involving the whole brain. Focal lesions can "stroke out"
particular functions, causing paralysis, speech deficits, or sensory
loss, but the entire brain must be involved for memory deficits,
disorientation, or obsessive behavior to occur.

Clearly, what was going on in the bicycle boy was amore than a simple
radiculitis: in which only a single nerve root would be affected.

By 1982, physicians in Connecticut had been alerted to the possibility
of Lyme arthritis, and the boy's first attack of knee pain had been
treated with a form of tetracycline. But two years later, when the boy
started to withdraw from life, started to become a behavior problem,
his physicians made no connection between his psychiatric symptoms and
his earlier episodes of arthritis.

"Lyme arthritis" was a disease of the joints or, at most, of the skin
and the joints: nobody had any basis for suspecting a connection
between the knee and brain disease - except perhaps for Andrew Pachner.

... Pachner had begun to uncover neurological symptoms and findings in
his Lyme arthritis patients. Another neurologist, Louis Reik, who had
preceded Pachner in the arthritis clinic, had passed on his suspicions
that the Lyme patients might have more than simple radiculitis
complaints. But it was up to Pachner to push ahead with his
observations.

Pachner connected the symptoms of the European patients to the new,
more diverse symptoms he was seeing in the Yale clinic. Reading through
the chart of that 12-year-old boy, Pachner began to get excited.

Could this boy have an infection that affected not just his knee but
his brain as well? The organism identified as causing Lyme arthritis
was a spirochete. Syphilis was a spirochete, and what syphilis could do
to a brain was well-known. It could cause dementia, bizarre pain
syndromes, a whole variety of symptoms so diverse that medical students
are taught to think of syphilis as the "great imitator".

Syphilis mimics many diseases because it can affect so many organs:
heart, brain, joints, nerve, eye. Wherever blood goes, syphilis can go.
Syphilis can cause a vasculitis of the small blood vessels in the
brain, the eye, almost anywhere. Could this new spirochete, this
borrelia burgdorferi, be as strange and protean in its manifestations
as the "great imitator" itself?

Could it be, thought Pachner, that this bicycle boy has Borrelia in his
brain?

If the spirochete that causes syphilis can enter the body through
genital tissues, multiply, migrate to small branches of the vascular
tree, migrate through the thin blood-vessel walls, and set up house in
the brain and nervous tissue, and in heart tissue and aorta, was it so
farfetched to believe that the Lyme spirochete might do something
similar?

Might it enter the body through a break in the skin caused by an insect
bite, the way malaria does, enter the blood stream, and multiply first
in a knee joint causing arthritis, and then wreak havoc years later in
the brain, as syphilis has been known to do?

Not having an answer, not having solid evidence or similar cases,
Pachner could not voice his suspicions to the boy's parents. He spoke
instead to the psychiatrists and asked them to transfer the boy to Yale
- New Haven Hospital. The parents were told simply that there was a
chance the therapy at Yale could help their son. They were willing to
try anything.

When the boy arrived at the hospital, he was taken to the neurological
ward . Pachner met his parents and explained that he believed there
might be a connection between their son's previous bouts of arthritis
and the problems that had landed him on the psychiatric ward. But
Pachner could make no promises--they were in uncharted waters.

The boy's parents did not know what to say. Their son's strange course
had been so baffling, their odyssey through the psychiatric wards so
bizarre, they could accept anything. They had no choice but to hope
that Andrew Pachner was correct.

On the neurology ward, Pachner did a lumbar puncture on the boy,
inserting a needle into the midline of his back, passing it between the
vertebral bones to the fluid-filled sac called in which the spinal cord
floats. Examining the fluid, called cerebrospinal fluid, or CSF,
Pachner noted a profusion of immune cells called lymphocytes.

Now he knew he had something. Patients in Europe who had neurological
symptoms following arthritic disease showed similar findings in their
cerebrospinal fluid.

Those lymphocytes might be the marker for the presence of the Borrelia
spirochete. Pachner ordered an intravenous line started on the boy and
20 million units of penicillin to be infused daily for fourteen days.

There was no reason to expect sudden response or improvement. If
Pachner was right, if the boy's current depression and compulsive
behavior were attributable to a brain infection with the spirochete
Borrelia, then the initial infection dated back two years, to his first
episode of arthritis. A long standing, deep-seated infection like that
could not be expected to be resolved overnight.

But the response was dramatic. Within days of the initiation of
therapy, Pachner recalls, "his behavior changed."

The parents were speechless . Even now, Pachner finds it difficult to
describe the sensation of watching those first changes in the boy.

"It was like-" Pachner searches for a word, shakes his head, then
finally says, "a fairy tale. That's all you can say ."

The boy was discharged. Pachner watched him leave with his parents. Two
weeks later, the boy arrived with his parents at Pachner's clinic. He
had gained weight, but more important, he was talking again, was more
outgoing, and had gone back to school.

Within months the boy was back playing soccer and he was doing his
homework. The transformation, or the reclamation, was complete. He was
back to normal.

In the process, the understanding of the disease that had been called
Lyme arthritis had expanded. The disease was no longer limited to the
joints. It would henceforth be called Lyme disease, a disease of many
organs, including the brain. IT WAS THE NEW GEAT IMITATOR.

Pachner has reported this new disease in many guises:

A 21-year-old man with a history of violent outbursts, confusion, and
wild laughing was thought to have a herpes-virus infection of his
brain; treated for Lyme disease, he returned to normal.

A 55-year-old woman who had gone to her doctor with a facial droop was
cured after a diagnosis of Lyme disease led to early treatment with
intravenous penicillin.

A 37-year-old man with fatigue, a sore throat, joint and muscle pains,
and facial-muscle paralysis who was thought to have multiple sclerosis
was found to have Lyme disease, and all symptoms resolved.

A 61-year-old man with double vision who was thought to have a brain
tumor was treated for Lyme disease with only partial improvement,
probably because his disease was too advanced to be cured.

And a 6-year-old girl suffering from headaches, knee pain, and tingling
in her toes -- and later from vertigo and staggering -- was apparently
[!!!] cured after treatment for Lyme disease followed positive studies
of her blood and cerebrospinal fluid.
Pachner thinks about the bicycle boy and says he was just one of many
cases. His eyes widen: "There are so many ways it can present. And
there are so many ways it presents that look like bad diseases, that
when you identify it and your reverse it - YOU FEEL LIKE GOD!" [ a
dangerous feeling ] ...
[Dr. Pachner] speaks of the subtle differences among the various
strains of the spirochete that may cause subtle differences in the
damage, the signs, and symptoms of the disease. In his laboratory, he
is getting to know the spirochete, or the "bug," as he calls it. He is
fascinated by the mysteries:

Deer, for example, do not get sick, although they harbor large numbers
of Borrelia organisms. Why? "Host defenses," Pachner says. [How do we
know that the deer do not have long-term problems, if they live long
enough?] ...

He seems driven by the will to know. He was working on his studies of
the Lyme disease patients while he was living the impoverished life of
a neurology resident, moonlighting like mad ...

Pachner left Yale for Georgetown in 1987, following Johnathan Pincus,
the Yale professor of neurology who had been appointed chairman of the
neurology department at Georgetown. Pinicus, author of the classic
textbook Behavioral Neurology, was able to attract Pachner offering lab
space and freedom to pursue his research interests.

Pachner shows me around his laboratory, of which he is proud. "I
remember how scarce lab space was at Yale, how people doubled up and
scraped by."

The lab Pachner has at Georgetown would have been considered a land of
milk and honey at Yale. Several technicians work for him, and they are
busy with lab chores. He has set up an assay for the Lyme antibodies,
and a technician shows him some "runs."

The blood samples are sent in from local physicians, and some test
positive: There is Lyme disease in the Washington area.

Although Lyme disease is known to occur in may countries, particularly
in Europe, and in 45 states in this county, the Mid-Atlantic and New
England states have an especially high infestation rate.

The tick that carries the disease, Ixodes dammini, [Ixodes scapularis]
clings to deer, field mice, and even dogs. Because the ticks are so
small, their human victims are often unaware of having played host to
this blood sucker, which may cling for four to six days to an
unsuspecting body.

In endemic areas such as certain parts of New England and Washington,
any patient who walks into the doctor's office with one side of his
face drooping in the classic manner of Bell's palsy should be suspected
of Lyme disease. And Bell's palsy is only one common neurologic
complication.

Since Pachner's studies called attention to the many sites that may be
inhabited by the spirochete, attention has also been focused on heart
lesions, which vary from direct attack on the heart-muscle wall -
myocarditis - to an attack penetrating every layer of the heart from
the inner lining through the heart walls to its coverings -pancarditis.

Patients with Lyme disease can show up at the doctor's office with
anything from severe chronic fatigue to arm pain to a variety of
palsies to arthritis and skin rashes. Erroneous diagnoses of dementia,
multiple sclerosis, psychiatric disease, and arthritis are common, so
closely can the great imitator mimic the symptoms of other illnesses.

The diagnosis can be difficult even when the physician suspects Lyme
disease. In Pachner's laboratory at Georgetown, blood, spinal fluid, or
joint fluid from patients with Lyme disease often fails to yield
positive cultures for the spirochete, which is difficult to keep alive
outside the body.

While Pachner's laboratory has the highest-quality technicians and
antiserums, only about half the patients are positive for the antibody
to the B. burgdorferi spirochete early in the course of the disease.
And, if the patient happens to be treated with an antibiotic before the
diagnosis is made, the antibody test may turn negative while living
spirochetes are still reproducing inside the body.

Making matters worse, antibody tests for Lyme disease may be falsely
positive in patients who have no Lyme spirochetes but who have instead
syphilis or other disease. Special antibody tests have to be done to be
sure the doctor is not dealing with a "false positive," in which the
test is positive but the patient has no Lyme disease.

Questions have been raised about the wisdom of any pregnant woman in an
endemic area such as Washington venturing into wooded areas during tick
season. Late spring and early summer are the peak times for the bites
that leave the hallmark skin rash, but patients can be infected on any
warm day of any month.

There is still no clear evidence about how much risk Lyme disease poses
to a developing fetus, but in the absence of hard data, may physicians
point to the concept that Andrew Pachner's studies implied: This
spirochete behaves in many ways like syphilis, infiltrating along blood
vessels. With syphilis as a model, few physicians feel comfortable
about the risks for mother and child infected with Lyme disease.

With its many parks running through the heart of the city, with the
C&O-Canal running into the heart of Georgetown, Washington is an area
in which the country laps up to the front door of suburban and urban
dwellers. Deer are common along the canals far into town as Glen Echo
and Brookmont on the Maryland-District line and, in Virginia, along the
George Washinton Parkway almost to Rosslyn.

Over the coming years, as Washington physicians become more aware of
its many guises, more and more cases of Bell's palsy, dementia,
fatigue, and arthritis will prove to be Lyme disease.

And there may even be a few boys who have withdrawn from friends and
families-boys who are languishing on psychiatric wards-whose blood or
spinal fluid will wind up in Andrew Pachner's lab, registering
positive. [ What about those who "register" negative on the ELISA or
Western Blot?]''

http://www.centurytel.net/tjs11/bug/ewald4.htm
Kathleen

a_weisman@yahoo.com wrote:
> eugeneshapiroisapig wrote:
> > I've been wondering if anyone had done a study comparing the
> incidence
> > and frequency of people going postal with the emergence of lyme
> > disease. By going postal, I mean a guy gets pissed off, gets a
bunch
> of
> > guns and goes apeshit. I would include individuals who commit
> multiple
> > homicide shooting sprees pretty much on impulse. Has the incidence
> > increased since the mid 70's? When the guy at Texas in 1960's
killed
> > all those people from the clocktower, they later found a
glioblastoma
> > multiforme tumor the size of a walnut compressing one amygdala of
his
> > brain. He left notes asking for an autopsy to determine the cause
of
> > his severe headaches and his uncontrolled impulse to kill people.
Of
> > course he had previously seen a doctor for these problems and they
> sent
> > him to a psychiatrist. I guess some things never change.
> > I wonder if any of our recent postals have felt ill,
fatigued,
> > etc. before going crazy. Maybe they get sick with LD, mess up their
> > jobs, get fired, and then freak out and kill in blind rage. Maybe
> not.
> > Who knows. Just wondering. Is there an epidemic of people going
> postal
> > in the US? If so,what might be contributing to it?
>
>
> Fallon testified in the attempted axe murder case for a Lyme patient.
> Dr Bransfield has wanted to do studies on road rage and domestic
> violence and Lyme.
>
> >From a "political" standpoint I don't think this helps us but the
facts
> are the facts.
>
> Of course Bransfield goes a bit overboard I think. He thinks
EVERYTHING
> is Lyme. In fact he wrote an article in the Lyme Alliance newsletter
> saying that Lyme explains homosexuality. Seriously he did. Another
> doctor (Katzel in San Fran) resigned from the LA board over it.
>
> ***********************************************************
>
> A.J. Feb 1 2001, 2:01 pm show options
>
> Newsgroups: sci.med.diseases.lyme
> From: A.J. <livegh...@snet.net> - Find messages by this author
> Date: Thu, 01 Feb 2001 17:02:53 -0500
> Local: Thurs, Feb 1 2001 2:02 pm
> Subject: "The Tick Defense "
> Reply to Author | Forward | Print | Individual Message | Show
original
> | Report Abuse
>
> I have mixed feelings about posting this,but I know this case has
been
> mentioned here, so here it is. We have to be aware of both good and
> bad news. The sad thing is that Lyme rage is very real, but the use
of
> it as a criminal defense is the sort of thing that will get the whole
> issue misunderstood and scorned if it gets noticed by the press.
> Amy in CT
>
>
>
http://dailynews.yahoo.com/htx­/hsn/20010131/hl/the_tick_defe­nse_1.html
>
>
>
> Wednesday January 31 03:05 AM EST
> The Tick Defense
>
>
> By Neil Sherman
> HealthScout Reporter
>
>
> MONDAY, Jan. 29 (HealthScout) -- A young man about to stand trial on
> charges that he axed his neighbor is offering an unusual
> alibi: He says Lyme disease made him do it.
>
>
> In January 1996, 17-year old Michael Griffin of Madison, Conn.,
> allegedly attacked Jim Disston with a medieval-style ax after he
> had knocked at his neighbor's door. Diston fought Griffin off and
> called the police, who later found the assailant in a closet,
> wielding a knife.
>
>
> A deer tick had bitten Griffin in 1991, giving him Lyme disease. His
> attorneys are planning to use the infection and its suggested
> neurological damage as his defense when Griffin comes to trial next
> month.
>
>
> It's entirely possible, says Dr. Virginia T. Sherr, a physician in
> private practice in Holland, Pa. "I live in an area highly endemic
> with Lyme disease, and I'm seeing a flood of mental symptoms from
> people who have unsuspected, undiagnosed Lyme disease.
> And neurological Lyme disease causes a lot of rage, like road rage or
> other forms of unprovoked rage."
>
>
> Lyme disease, caused by the Borrelia burgdorferi bacterium and
> transmitted by tiny deer ticks, has been reported in nearly
> every state and throughout Europe and Asia. It begins as a skin rash,
> and early symptoms often are flu-like, including fatigue,
> headache, fever, muscle stiffness and joint pain. Now the most common
> insect-borne infection in the United States, Lyme disease
> is increasing in incidence and geographic spread.
>
>
> I>Borrelia burgdorferi "puts out toxins which causes an inflammation
> of the blood vessels of the brain," Sherr explains. "And
> usually what I hear, when a person calls, is that there has been a
> sudden personality change in a family member. A person who
> was normally mild all of the sudden overreacts out of the blue, a
> sudden, almost explosive outpouring of rage right from the
> subconscious mind.
>
>
> "The spirochete attacks the nervous system," Sherr continues. "Lyme
is
> primarily a disease of the nervous system, though the men
> in the ivory tower often believe it's a joint problem. And as a
> disease of the nervous system it is known to cause
> personality/cognitive/emotiona­l/nervous system disease."
>
>
> Another expert dubious
>
>
> But if Lyme disease caused Griffin's behavior, "he would probably be
> the first such patient ever recorded," says Dr. Louis Reik,
> Jr., a professor of neurology at the University of Connecticut
Medical
> Center in Farmington. Reik says he knows of no other
> cases of violent behavior attributed to Lyme.
>
>
> "The most common thing it causes is a facial palsy, a temporary
> paralysis of one side of the face, and that gets better even without
> treatment," Reik says. "The next most common problem is meningitis,
an
> inflammation of the spinal fluid and the covering of the
> brain, which is often accompanied by a paralysis of the cranial
nerves
> and the spinal nerves."
>
>
> Reik says only when the disease is not treated do cognitive problems
> develop. "And that's usually pretty mild -- problems in
> concentrating, memory, paying attention -- and that usually goes
along
> with fatigue."
>
>
> Seldom are there cases of neurological damage, Reik adds. "Very, very
> rarely there is a more severe form of involvement.
> Basically what happens is that there is a meningitis and the little
> blood vessels inside the brain get inflamed and you get little
stroke,
> which do damage to the substance of the brain."
>
>
> "But that's very uncommon in this country," Reik says. "It's more
> common in Europe."
>
>
> Disston, the victim, declined to comment on the particulars of
> Griffin's defense. But, he says, "I just want him to be held
> accountable."
>
>
> The Lyme Disease Foundation is standing at arm's length of this case,
> refusing to comment about it.
>
>
> What To Do
>
>
> For more on Lyme disease, check out information provided by the
> National Institute of Allergy and Infectious Diseases and by
> Pfizer, the pharmaceutical company.
>
>
> Or, you may want to read previous HealthScout articles on Lyme
> disease.

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