Re: Study reveals new data on how lyme disease is spread



Jeepers,

Barbour published about that in 1986.

It was the state of the art in 1975.

The only reason we can't find "Lyme"
in the blood, is because we use primers
intended to not find it. Klempner refused
to tell me which primers he used in his
Chronic Lyme "study."

BSK does not support all borrelial
spirochetes, and that is intentional.

This we also learned from the pharming
of OspA out of E. coli.

It isn't possible that these guys are
so ignorant. "The Biology of Borrelia
Species" had over 250 references in it
some going back to the 1920s.

And that was published by Barbour in
1986. The report by Steere that same
year on what is the serology of Lyme,
looked like Steere knew absolutely
nothing about spirochetes. Or for
that matter, infections.

Kathleen

Martijn wrote:
> http://tinyurl.com/dggob
> Public release date: 2-May-2005
>
> *Study reveals new data on how lyme disease is spread*
>
> New York Medical College researchers publish new findings on the
spread
> of lyme disease bacteria
>
> The results of a five-year study, published this week in the Annals
of
> Internal Medicine by researchers at New York Medical College, reveal
> intriguing new data on the spread of the Lyme disease bacteria
through
> the blood stream. The ability to find the Lyme spirochete--the
> tick-borne agent responsible--in the blood is itself an achievement
> because existing methods of culturing blood were not sensitive enough
to
> detect its presence until College researchers developed a new
technique,
> which they used in the study.
>
> Leading the study was Gary P. Wormser, M.D., professor of medicine,
> director of the Division of Infectious Diseases and vice chairman of
the
> Department of Medicine. "If Lyme disease stayed in the skin it would
be
> a completely different and rather inconsequential infection--but it
> doesn't," Dr. Wormser explained. "The causative agent of Lyme disease

> can spread from its entry point at the tick bite site through the
blood
> to distant sites such as the brain, heart, and joints. This study
> answers questions that have never been answered before and raises
others
> that will likely stimulate future studies on Lyme disease."
>
> The study followed 213 initially untreated adults with erythema
migrans,
> the tell-tale bull's eye rash that is the most common clinical
feature
> of Lyme disease, to determine when does blood stream invasion occur,
how
> many patients experienced blood stream invasion and who is most
> vulnerable. Blood stream invasion occurred in 93 (43.7 percent) of
> patients in the study, who also were more often symptomatic (89.2
> percent vs. 74.2 percent) and more likely to have multiple erythema
> migrans lesions (41.9 percent vs. 15.0 percent) than the 120 patients

> without blood stream invasion. However, some patients had no
tell-tale
> symptoms at all, making the presence or absence of blood stream
invasion
> impossible to predict with certainty based on clinical features
alone.
> Younger patients and those with a prior history of Lyme disease were
> somewhat protected.
>
> Dr. Wormser and his colleagues concluded that, "The high rate, early
> onset, and prolonged duration of risk for blood stream invasion
probably
> explain why untreated patients with erythema migrans often develop
> complications distant from the tick bite location. Older patients
> without a past episode of Lyme disease are at particular risk."

.


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