Re: BROOKHAVEN: "It's the perfect stealth bacteria," says one frustrated physician. He's talking about Borrelia burgdorferi, the bacterium that causes Lyme disease. This illness, which is often mistaken for diseases ranging from multiple sclerosis to Lupus, can inflict excruciating headaches and muscle pain, affect the brain and nervous system, attack major organs, and inflame joints.




overman74@xxxxxxxxxxx wrote:
> So, how did OspC get to be king anyway. If the strain(s) I got did not
> express this, per the missing band on my western blot, then it isn't
> much good as a diagnostic element for those strains.
>
> I know, go see your website. But it is hard to find things there
> sometimes. I suppose the explanation is in the Dressler stuff.


OspC is the neurotropism antigen and also is the
thing expressed in the on the bug in the tick when feeding.

These antigens have specificities for tissue types.

One would imagine there is something on red blood cells
that is OspC friendly.

On my website, on the homepage, almost at the bottom, is a picture of
OspC next to Barbour's text about neurotropism. Dattwyler calls it an
invasiveness characteristic. Dattwyler and Luft wanted to make an
OspC vaccine.
That was always a much more reasonable choice than OspA. I have no
idea
why they wanted to go ahead with OspA, when they knew it showed up
later, and
was associated with knees more than anything, other than the patent was
owned
by Yale, and they insist this is an arthritic disease, despite the
abundance of
evidence that this is a borreliosis. Steere really was *DUMB* in the
early years
as you can read from his first serology survey. He truly is not very
smart.

Perhaps he was chosen by the bioweaponeers at Plum Island and Yale
CIA/CDC because he is a Dr. Bumble.

http://actionlyme.org/
"The association of borreliae with ganglia or other nerve tissue has
also been noted in other tick species (102) and in lice (116, 126).
The propensity for borrelia to go to the brain of infected mammals (see
below) suggests that the relationship between these spirochetes and
neural tissues is not trivial. Further study of this attraction and
the interaction that follows may reveal the basis for the significant
nerve and brain involvement in Lyme borreliosis (201, 206, 233; R.
Ackermann B. Rehse-Kupper, and E. Gollmer, Sentralb Bakteriol.
Mikrbiol. Hyg., in press; A. R. Pachner and A. C Steere, Zentralb.
Bakterio. Mikrobiol. Hyg. Ser.. A, in press)."--- The Biology of the
Borrelia species, AG Barbour, SF Hayes, 1986 OspC is
associated with neurotropism.

.


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