Re: NEWS: Mark Klempner and The Science.

On Jun 8, 4:07 pm, the 3rd Man <derdrittemann2...@xxxxxxxxx> wrote:
On Jun 8, 12:59 pm, Mort Zuckerman <morph...@xxxxxxxxx> wrote:

"Moreover, antibiotic treatment does not preclude isolation of viable
spirochetes
from patients. For example, B. burgdorferi has been isolated from
skin, synovial
fluid and cerebrospinal fluid of patients who received antibiotic
treatment for
Lyme disease...

SO?

That doesn't necessarily mean that the disease has not been "CURED".

Depends on what you mean by "cured"...how you define it.

If you are, I would think, relieved of symptoms...I would guess most
people would believe themselves "cured"...even if a few leftover
spiros remain.

Unless the body's immune response is now totally and completely
ineffective...wouldn't one expect that the immune system could handle
the mop-up of remaining spirochetes?

"CONCLUSION: Ceftriaxone fails to cure Lyme Disease due to the
intracellularity
of the spirochetes."

NO.

Show us where Klempner said that...(and PLEASE...remind us here how
you NEVER "lie").

And you REALLY should NOT make up things and FALSELY attribute them to
another.

It's EXTEREMELY DISHONEST...and terribly misleading.

Fibroblasts protect the Lyme disease spirochete, Borrelia burgdorferi,
from ceftriaxone in vitro.
Georgilis K, Peacocke M, Klempner MS.

Department of Medicine, New England Medical Center, Boston,
Massachusetts.

The Lyme disease spirochete, Borrelia burgdorferi, can be recovered
long after initial infection, even from antibiotic-treated patients,
indicating that it resists eradication by host defense mechanisms and
antibiotics. Since B. burgdorferi first infects skin, the possible
protective effect of skin fibroblasts from an antibiotic commonly used
to treat Lyme disease, ceftriaxone, was examined. Human foreskin
fibroblasts protected B. burgdorferi from the lethal action of a 2-day
exposure to ceftriaxone at 1 microgram/mL, 10-20 x MBC. In the absence
of fibroblasts, organisms did not survive. Spirochetes were not
protected from ceftriaxone by glutaraldehyde-fixed fibroblasts or
fibroblast lysate, suggesting that a living cell was required. The
ability of the organism to survive in the presence of fibroblasts was
not related to its infectivity. Fibroblasts protected B. burgdorferi
for at least 14 days of exposure to ceftriaxone. Mouse keratinocytes,
HEp-2 cells, and Vero cells but not Caco-2 cells showed the same
protective effect. *** Thus, several eukaryotic cell types provide the
Lyme disease spirochete with a protective environment contributing to
its long-term survival. ***
http://www.ncbi.nlm.nih.gov/pubmed/1634816?ordinalpos=2&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum


It only takes one spirochete to start the "disease."

In addition, the bad guys have all referenced this URI report:
http://www.actionlyme.org/IDSA_CYST_VIABLE.htm

And as usual, the name of the disease is Relapsing Fever
since it is incurable.

http://www.actionlyme.org/RICOCHRON.htm

AS USUAL, I *NEVER* *LIE.*

.