Re: Does calcium citrate reduce stomach acid?


"Pizza Girl." <nos.pam@xxxx> wrote in message
news:1127528867.e1658d9d86985c55e1460177ad620902@xxxxxxxxxxx
> How could people get hypocalcemia when calcium is so readily available for
> green vegetables and other foods.
>
> I guess dairy consumption can lower absorption of calcium and since the
> market propaganda started 40 years ago people can hurt themselves with too
> much dairy.

Am J Clin Nutr. 2005 Sep;82(3):523-30. Related Articles, Books, LinkOut

Dairy consumption is inversely associated with the prevalence of the
metabolic syndrome in Tehranian adults.

Azadbakht L, Mirmiran P, Esmaillzadeh A, Azizi F.

Endocrine Research Center, Shaheed Beheshti University of Medical Sciences,
Tehran, Iran.

BACKGROUND: Although previous studies showed some benefits from dairy
consumption with respect to obesity and insulin resistance syndrome,
epidemiologic data on the association between dairy intakes and metabolic
syndrome are sparse. OBJECTIVE: The objective was to evaluate the relation
between dairy consumption and metabolic syndrome in Tehranian adults.
DESIGN: Dairy consumption and features of metabolic syndrome were assessed
in a population-based cross-sectional study of 827 subjects (357 men and 470
women) aged 18-74 y. Metabolic syndrome was defined according to guidelines
of the Adult Treatment Panel III. Multivariate logistic regression adjusted
for lifestyle and nutritional confounders was used in 4 models. RESULTS:
Mean (+/-SD) consumption of milk, yogurt, and cheese was 0.7 +/- 0.2, 1.06
+/- 0.6, and 0.9 +/- 0.3 servings/d, respectively. Subjects in the highest
quartile of dairy consumption had lower odds of having enlarged waist
circumference [odds ratio (OR) by quartile: 1, 0.89, 0.74, 0.63; P for trend
< 0.001], hypertension (OR by quartile: 1, 0.88, 0.79, 0.71; P for trend <
0.02), and metabolic syndrome (OR by quartile: 1, 0.83, 0.74, 0.69; P for
trend < 0.02). The values of ORs became weaker after further adjustment for
calcium intake. CONCLUSION: Dairy consumption is inversely associated with
the risk of having metabolic syndrome. It seems that this relation is
somewhat attributed to calcium.

PMID: 16155263 [PubMed - in process]

Clin Calcium. 2005 Feb;15(2):255-60. Related Articles, Books, LinkOut
[Magnesium and hypertension]

[Article in Japanese]

Rosanoff A.

Independent Scholar (Hawaii, U.S.A).

Magnesium status has a direct effect upon the relaxation capability of
vascular smooth muscle cells and the regulation of the cellular placement of
other cations important to blood pressure - cellular sodium:potassium (Na:K)
ratio and intracellular calcium (iCa(2+)). As a result, nutritional
magnesium has both direct and indirect impacts on the regulation of blood
pressure and therefore on the occurrence of hypertension. Hypertension
occurs when cellular Na:K ratios become too high, a consequence of a high
sodium, low potassium diet or, indirectly, through a magnesium deficient
state which causes a pseudo potassium deficit. Like wise, magnesium
deficiency alters calcium metabolism, creating high iCa(2+), low serum
calcium and low urinary calcium states even when calcium intake is adequate.
High iCa(2 + ) and high cellular Na:K ratio both occur when cellular
magnesium becomes too low and the Mg-ATP driven sodium-potassium pump and
calcium pump become functionally impaired. High iCa(2+) has several
vasoconstrictive effects which lead to hypertension, an indirect result of
low magnesium status. Dietary calcium is directly proportional to dietary
magnesium. Serum magnesium does not reflect true magnesium status as do
intracellular magnesium measurements. Several studies on the effect of
calcium on blood pressure need these added considerations of magnesium
status to fully understand the impact of the Mg:Ca ratio as the primary
cause of hypertension and other aspects of Syndrome X. Magnesium
supplementation above 15 mmol per day are required to normalize high blood
pressure in unmedicated hypertensive patients while 15 mmol per day will
lower high blood pressure in patients treated with anti-hypertensive
medications. In most humans, healthy blood pressure depends upon a balance
of both Na:K and Mg:Ca ratios at both cellular and whole body levels which,
in turn, require adequate, long-term intakes of nutritional magnesium. The
knowledge that low magnesium causes imbalance in both cellular and
physiological calcium widens our view of the studies showing hypertensives
have abnormal calcium metabolism.

Publication Types:
Review




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