Re: Flax oil, kefir, yogurt producing no immune system benefits

Wed, 25 Jan 2006 14:46:31 +0200 in article
<fsret11k1pong3rr57b4am81a53vilh9g9@xxxxxxx> Matti Narkia <narkia@xxxxxxxxx>
wrote:

>Tue, 24 Jan 2006 05:23:56 GMT in article
><MRiBf.1454$1n4.872@xxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxx> "Knack"
><zymatik@xxxxxxxxxxxxxxx> wrote:
>
>>
>>> You don't seem to be doing terribly well. I cannot remember when I last
>>> had
>>> cold or flu, must have been years ago. And last time i had cold or flu
>>> which
>>> lasted 10 days or more was in 1988. Perhaps you do need to take more
>>> selenium, zinc, vitamin C, astragalus, propolis, garlic, etc., etc. ...
>>> ;-).
>>
>>Well, I can't see taking larger quantities of the routine pills that I
>>already take, but perhaps I could take those special extracts such as
>>astragalus, echinacea, etc. the day before I get on a plane, and then
>>continue taking them for a few more days. But if those timely special
>>extracts do the trick, then what good are all my regular routinely taken
>>pills?
>>
>Are you a clairvoyant? Can you tell in advance, when you are going to get
>infection? Of course not, but even if you did, the things you take may need
>time to "do the trick". And suppose you have a Th1/Th2 imbalance with Th2
>dominance, which is fairly common. To avoid problems you would then perhaps
>need to take something continuously to restore and retain the balance.
>Roughly speaking the Th1 cells are responsible for the cell mediated
>immunity and the Th2 cells for humoral immunity. If you have Th2 dominance,
>you have problems with cell mediated immunity. The easiest way to determine
>the status of your cell mediated immunity is to take the delayed
>hypersensitivity test, see for example
>
>Delayed-type Hypersensitivity Test (DTH): Information From Answers.com
><http://www.answers.com/topic/delayed-type-hypersensitivity-test-dth>
>
>Delayed hypersensitivity skin test
><http://health.enotes.com/medicine-encyclopedia/delayed-hypersensitivity-skin-test>
><http://www.healthatoz.com/healthatoz/Atoz/ency/delayed_hypersensitivity_skin_test.jsp>

Some additional links:

Immune Defense against Microbial Pathogens
<http://textbookofbacteriology.net/immune.html>

"Cell-mediated immunity (CMI) is the type of immunity that is
mediated by specific subpopulations of T-lymphocytes called
effector T cells. In non immune animals precursor T cells (pT
cells) exist as "resting T cells". They bear receptors for
specific antigens. Stimulation with Ag results in their
activation. The cells enlarge, enter into a mitotic cycle,
reproduce and develop into effector T cells whose activities
are responsible for this type of immunity. They also develop
into clones of identical reactive T cells called memory T
cells.

The biological activities of the antibody-mediated and cell-
mediated immune responses are different and vary from one type
of infection to another. The AMI response involves interaction
of B lymphocytes with antigen and their differentiation into
antibody- secreting plasma cells. The secreted antibody binds
to the antigen and in some way leads to its neutralization or
elimination from the body. The CMI response involves several
subpopulations of T lymphocytes that recognize antigens on the
surfaces of cells. TH cells respond to antigen with the
production of lymphokines. The distinction between TH1 and TH2
is based on their lymphokine profiles. TH2 cells have
previously been referred to as T helper cells because they
provide lymphokines (e.g. IL-2 and IL-4) which activate T
cells and B cells at the start of the immune response. TH1
cells were formerly known as delayed type hypersensitivity
cells (TDTH) because of their role in this allergic process.
TC cells or cytotoxic T lymphocytes (CTLs) are able to kill
cells that are showing a new or foreign antigen on their
surface (as virus-infected cells, or tumor cells, or
transplanted tissue cells)."

eMedicine - Hypersensitivity Reactions, Immediate : Article by Miriam K
Anand, MD
<http://www.emedicine.com/med/topic1101.htm>

"Pathophysiology: Immediate hypersensitivity reactions are
mediated by IgE, but T and B cells play important roles in the
development of these antibodies. CD4 cells or helper T (TH)
cells have been divided into 2 broad classes based on the
cytokines they produce.

TH1 cells produce interferon gamma, interleukin (IL)?2, and
tumor necrosis factor-beta and promote a cell-mediated immune
response (eg, delayed hypersensitivity reaction). TH2 cells,
on the other hand, produce IL-4 and IL-13, which then act on B
cells to promote the production of antigen-specific IgE.
Therefore, TH2 cells play an important role in the development
of immediate hypersensitivity reactions, and patients who are
atopic are thought to have a higher TH2-to-TH1 cell ratio.
Interestingly, the cytokines produced by TH1 cells
(specifically interferon gamma) seem to diminish the
production of TH2 cells. "

Delayed Type Hypersensitivity
<http://dermatology.cdlib.org/DOJvol5num1/reviews/black.html>

Cell-Mediated Immunity
<http://www.macses.ucsf.edu/Research/Allostatic/notebook/cellimmunity.html>


--
Matti Narkia
.

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