Re: Another lesson from "Eskimos."
- From: "MikeV" <mavidler@xxxxxxxxxxxxx>
- Date: Sun, 29 Mar 2009 12:36:25 -0400
An interminable walking hypothesis condemned for all eternity to troll for scientific absolution. How sad is that?
" ... It ain't ignorance that causes all the trouble in this world. It's the things people know that ain't so."
Edwin Armstrong
Mike V
Celebrating 11 years and counting on fish oil, and vitamin D3.
<monty1945@xxxxxxxxx> wrote in message news:bb67a7db-e962-47b9-9133-664d4a9fc184@xxxxxxxxxxxxxxxxxxxxxxxxxxxxxxx
On page 192 of the book "Diet and Health" (by the Nation Research
Council): "Greenland Eskimos have been
cited as an example of a population with a high intake of fats and
oils
from marine sources and very low rates of CHD (Bang and Dyerberg,
1980), but the epidemiologic data are insufficient to confirm this
assertion. In fact, the risk of CHD is unknown in this small
population, whose members live under harsh conditions and usually die
before middle age."
You can see this page via google books. Some have criticized my
citation of this, arguing that they died becauseo fo the "harsh"
lifestyle, but these are the same people who then talk about the Masai
and other "primitive" peoples without mentioning the harsh lifestyle.
However, as other investigators have pointed out, when you are on an
omega 3 rich diet, you run the risk of bleeding to death from minor
blunt-force trauma and "bleeding strokes," and so their deaths at
early ages are (or were) consistent with what is known at the
molecular-level, and probably not related to an especially "harsh"
lifestyle. You can do some searches and find the evidence, such as:
Scand J Clin Lab Invest Suppl. 1982;161:7-13.
"A hypothesis on the development of acute myocardial infarction in
Greenlanders."
Dyerberg J, Bang HO.
Abstract/summary: Non-emigrated Greenlanders have a low incidence of
acute myocardial infarction (AMI), when compared with age- and sex
adjusted death rates for ischemic heart disease in western countries.
We find that Greenlanders have plasma lipid levels corresponding to
favourable risk factor levels for AMI. This can be attributed to their
diet, rich in n-3 polyunsaturated fat. This diet further supplies
eicosapentaenoic acid which influence platelet vessel wall function in
an antithrombotic direction. A high level of plasma-antithrombin-III,
raising the anticoagulant activity of the blood, in combination with a
genetically high activation threshold for the complement system may
further contribute to the resistancy against thrombo-embolic
disorders. Bleeding tendency, and susceptibility to infection
disorders may be the possible draw-backs. Our data are framed into a
hypothesis combining the indications of genetic predispositions and
the evidence of exogenous protective factors, inflicting a coherent
enhancement of nonsusceptibility to vascular ischemic catastrophies.
The point I've been making for years now is that when you look at the
evidence comprehensively (as I was trained to do in grad school),
there is only one reasonable explanation, and that is that Mead acid
should be in your cells, rather than omega 3s and/or omega 6. I have
experimented with this notion directly on myself since 2001, and have
seen no problems, only benefits, for my "essential fatty acid
deficient diet." That is how science is supposed to work, that is,
you test a hypothesis directly and determine if it is accurate. If it
is not, then it must be abandoned. Much of the biology-related
sciences today, however, seem to operate more like cults, or
religions, with authoritative leaders trying to make their followers
obey the dogma and never question it.
.
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- Another lesson from "Eskimos."
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