ID Society's Bogus Lyme Claims


Reporting Crime to the US Attorney's Office; Thomas Carson @ US
Attorney Kevin O'Connor's office in New Haven, CT, who has already been
given all this data, 3 years ago, and who did nothing about it
http://www.actionlyme.org/USDOJ_COMPLAINT_RICO.htm
As you can see from a comparison of the blots, Lyme is an inflammatory
disease only in certain people. The blots on the right are that of
people with a genetic predisposition to have an allergy reaction (see
NIAID- National Institute of ALLERGY and INFECTIOUS DISEASE).

I will demonstrate that the Infectious Diseases Society of America's
new guidelines on Lyme:
http://www.idsociety.org/Content/NavigationMenu/Practice_Guidelines/Standards_Practice_Guidelines_Statements/Standards,_Practice_Guidelines,_and_Statements.htm
are as big a lie as their last (which I demonstrated in Nov 2000
http://www.geocities.com/kmdickson0308/lyme-dilemma.html
That website has not been touched since Nov 2000.

========


1) Klempner determines that ceftriaxone does not kill all the
spirochetes:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=1634816&query_hl=47&itool=pubmed_docsum
"Thus, several eukaryotic cell types provide the Lyme disease
spirochete with a protective environment contributing to its long-term
survival."

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=8486939&query_hl=47&itool=pubmed_docsum
"These observations suggest that B. burgdorferi can adhere to,
penetrate, and invade human fibroblasts in organisms that remain
viable."

Klempner's Matrix-Metalloproteinases in the spinal fluid of Lyme
patients which he thinks are contributing to the dementia:
http://www.actionlyme.org/Retro_Klempnerization.htm
(You have to print out one jpg file at a time by selecting it first and
copying it to a Word document)
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=9466528&query_hl=52&itool=pubmed_docsum

Klempner later publishing that there are no valid markers of illness in
CNS Lyme patients and is an author of the IDSA guidelines:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=12821733&query_hl=64&itool=pubmed_docsum

=========

2) SIGAL AND QEEG:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_term=7554300%5BUID%5D
"Quantitative EEG, flash visual evoked potentials, auditory evoked
potentials to common and rare tones, and median nerve somatosensory
evoked potentials were obtained from 12 patients with active CNS Lyme
disease and from 11 patients previously treated for active CNS Lyme
disease. Abnormal QEEG and/or EPs were found in 75% of the active Lyme
disease patients and in 54% of the post CNS Lyme disease patients.
Three different types of neurophysiological abnormality were observed
in these patients including QEEG slowing, possible signs of cortical
hyperexcitability, and focal patterns indicating disturbed
interhemispheric relationships. In patients tested before and after
treatment QEEG and EP normalization was associated with clinical
improvement."

He later said we have fibromyalgia and that fibromyalgia is
catastophizing:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_term=sigal%20LH%5BAuthor%5D%20AND%20%28%22fibromyalgia%22%5BMeSH%20Terms%5D%20OR%20fibromyalgia%5BText%20Word%5D%29
He also said we're poisoning ourselves and our children, as a cause of
chronic Lyme disease:
http://www.actionlyme.org/MUNCHAUSENS.htm

3) Steere, Sigal, Rahn and Schoen report Lyme treatment fails in 1/2
the cases:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=3109144&query_hl=44&itool=pubmed_docsum

===========

4) Halperin and Quin as a neurotoxin
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Search&db=pubmed&term=quinolinic+acid+and+borrelia&tool=fuzzy&ot=quinolonic+acid+and+borrelia

TODAY (They don't know what Chronic Lyme is? see more below):
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=17029130&query_hl=66&itool=pubmed_docsum

Halperin is a contributor to the report that shows that Lou Gehrig's
Disease is associated with Lyme in 47% of the cases:
http://www.actionlyme.org/ALS_&_Lyme_47%25.htm

Why doesn't Halperin, an author of the new IDSA guidelines mention this
as an outcome of Lyme?

===============

5 &6) GFAp, Yale, Schoen, and Congenital Lyme:
Yale's Robert Schoen mentioning Gliosis (degradation of glial cells, or
the energy producing cells of the brain) as a serious outcome of
NeuroLyme:
http://www.annals.org/cgi/reprint/132/8/661.pdf

Who said what derogatory/mysogynistic remark and where it is published:
http://www.actionlyme.org/UN_PETITION.htm
"LYME PARANOIA"-- Robert Schoen, MD, Yale Rheumatology

Robert Schoen tells me I have "I don't know."
http://www.actionlyme.org/Schoen.htm while pregnant, having given Lyme
and probably ehlichiosis to my first two kids, and Yale knows Lyme
congentially confers, since Yale pathology contributed to this autopsy
of a dead congenital Lyme newborn
http://www.actionlyme.org/Congenital_Brain_Infection_of_Newborn_Resulting_in_Death.htm
See more reports about cogenital newborns, Lyme and maternal antibodies
- a report by Allen Steere.

The Publication of GFAp as a marker of brain/nerve degradation that
Schoen references:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_term=12938230%5BUID%5D

==============
6) NEOPTERIN (This report is not by a known US bad guy but is a valid
marker of infection)
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=7865624&query_hl=33&itool=pubmed_docsum

====
7) ANTIGANGLIOSIDE ANTIBODIES, Benach (who later published in the New
York Times that Steere was right, and Steere had the science on his
side, but that the patients don't, in a letter to the editor):
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=7558329&query_hl=32&itool=pubmed_docsum
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?itool=abstractplus&db=pubmed&cmd=Retrieve&dopt=abstractplus&list_uids=8410057
'Benach demonstrated that antibodies to gangliosides inhibit nerve
function in vitro.

Benach saying to the TIMES that we patients are idiots and Steere is a
genius:
http://query.nytimes.com/gst/fullpage.html?res=9F05EFDE173CF933A05756C0A9669C8B63

=====
8) ANTIPHOSPHOLIPID ANTIBODIES (LUPUS), Steere
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=3408508&query_hl=34&itool=pubmed_docsum
======

9) LYME AND ALL BORRELIOSES ARE PERMANENT BRAIN INFECTIONS
by Bergstrom, the business partner (See the US and European Espacenet
patent databases) of Alan Barbour, CDC EIS Officer and CDC Level IV
Bioweapons lab head at UCAL Irvine;
Alan Barbour says Lyme and Relapsing Fever permanently infect the brain
on his website:
http://www.ucihs.uci.edu/microbio/index.html?top.html&menu.html&facultyResearch/faculty/barbour.html
"Lyme disease and relapsing fever. These tick-borne infections are
notable for multiphasic antigenic variation through DNA recombinations
in the case of relapsing fever, the occurrence of chronic arthritis in
the case of Lyme disease, and invasion of and persistence in the brain
in the case of both diseases."

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_term=%28%22borrelia%22%5BMeSH%20Terms%5D%20OR%20borrelia%5BText%20Word%5D%29%20AND%20%28%22brain%22%5BMeSH%20Terms%5D%20OR%20brain%5BText%20Word%5D%29
Barbour says Lyme and Relapsing fever are nearly the same thing, and
both infect the brain, which used to be the culture media-rodent
brains, says Barbour in 1986:
http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pubmed&pubmedid=3540570

http://www.actionlyme.org
http://www.actionlyme.org/LYME_IS_A_PERMANENT_BRAIN_INFECTION.htm
All the other older reports where it was "well-known" that borrelia
permanently infect the brain
1: Microbes Infect. 2006 Jul;8(8):2213-9. Epub 2006 May 30.

Persistent brain infection and disease reactivation in relapsing
fever borreliosis.

* Larsson C,
* Andersson M,
* Pelkonen J,
* Guo BP,
* Nordstrand A,
* Bergstrom S.

Department of Molecular Biology, Umea University, SE-901 87 Umea,
Sweden.

Relapsing fever, an infection caused by Borrelia spirochetes, is
generally
considered a transient, self-limiting disease in humans. The present
study
reveals that murine infection by Borrelia duttonii can be reactivated
after an
extended time as a silent infection in the brain, with no bacteria
appearing in
the blood and spirochete load comparable to the numbers in an infected
tick. The
host cerebral gene expression pattern is indistinguishable from that of

uninfected animals, indicating that persistent bacteria are not
recognized by
the immune system nor cause noticeable tissue damage. Silent infection
can be
reactivated by immunosuppression, inducing spirochetemia comparable to
that of
initial densities. B. duttonii has never been found in any host except
man and
the tick vector. We therefore propose the brain to be a possible
natural
reservoir of the spirochete. The view of relapsing fever as an acute
disease
should be extended to include in some cases prolonged persistence,
a feature characteristic of the related spirochetal infections Lyme
disease and
syphilis. PMID: 16782384 [PubMed - in process]


VIJAY SIKAND SAYING LYME IS LATENT IN THE BRAIN LIKE SYPHILIS AT THE
FDA MEETING IN 1998 WHEN THEY WERE TRYING TO PASS OFF A BOGUS VACCINE:
http://www.fda.gov/ohrms/dockets/AC/98/transcpt/3422t1.rtf

"In particular, the specter of asymptomatic infection is something that
troubles me a great deal and troubles a great number of my colleagues
who need to treat Lyme disease. The obvious analogy with syphilis
infection with Treponema pallidus is there to consider. It is well
known that Borrelia burgdorferi indeed after asymptomatic infection can
lurk or secrete itself in certain areas of the body, perhaps the
central nervous system or perhaps the joint spaces, only to reappear
months or maybe years later in the form of late stages of illness which
are harder to diagnosis and treat..."

LYMErix was never demonstrated to prevent Lyme, since the testing for
Lyme is bogus:
http://www.fda.gov/ohrms/dockets/ac/01/slides/3680s2_11.pdf

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